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Fusion tyrosine kinases: A result and cause of genomic instability
E. T.P. Penserga,
T. Skorski
Nuclear Dynamics and Cancer (NDC)
Center for Immunology
Temple University
Research output
:
Contribution to journal
›
Review article
›
peer-review
62
Scopus citations
Overview
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Biochemistry, Genetics and Molecular Biology
Kinase
100%
Genome Instability
100%
Tyrosine
100%
Kinase A
100%
Nested Gene
28%
DNA Repair
28%
Protein
14%
Genetics
14%
Apoptosis
14%
Cell Cycle Checkpoint
14%
Adhesion
14%
DNA Damage
14%
DNA
14%
Stress
14%
Species
14%
Oxidative Stress
14%
Radiation
14%
Imatinib
14%
Methanesulfonic Acid
14%
Chromosomal Translocation
14%
Chimera
14%
S Cell
14%
Medicine and Dentistry
Genomic Instability
100%
Protein Tyrosine Kinase
100%
Protein Tyrosine Kinase A
100%
Gene
33%
DNA Repair
33%
Therapeutic Procedure
16%
Disease
16%
Cells
16%
Protein
16%
Cell Cycle Checkpoint
16%
Phosphotransferase
16%
DNA Damage
16%
Programmed Cell Death
16%
Tumor Cell
16%
Antiapoptotic
16%
Differentiation
16%
Oxidative Stress
16%
Reactive Oxygen Species
16%
Imatinib
16%
Stress
16%
Chromosome Translocation
16%
Chimera
16%
Double Stranded DNA
16%
Neuroscience
S Cell
14%
Chromosome Translocation
14%
Pharmacology, Toxicology and Pharmaceutical Science
Chromosome Translocation
16%